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Please use this identifier to cite or link to this item: https://saber.ucv.ve/jspui/handle/10872/15675
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dc.contributor.authorPaniz-Mondolfi, Alberto-
dc.contributor.authorSingh, Rajesh-
dc.contributor.authorJour, George-
dc.contributor.authorMahmoodi, Mandana-
dc.contributor.authorDiwan, A. Hafeez-
dc.contributor.authorBarkoh, Bedia A.-
dc.contributor.authorCason, Ronald-
dc.contributor.authorHuttenbach, Yve-
dc.contributor.authorBenaím, Gustavo-
dc.contributor.authorGalbincea, John-
dc.contributor.authorLuthra, Rajyalakshmi-
dc.date.accessioned2017-04-17T20:24:07Z-
dc.date.available2017-04-17T20:24:07Z-
dc.date.issued2015-
dc.identifier.issn1432-2307 (Electronic)-
dc.identifier.issn0945-6317 (Linking)-
dc.identifier.issn0945-6317 (Print)-
dc.identifier.otherDOI 10.1007/s00428-015-1718-7-
dc.identifier.urihttp://hdl.handle.net/10872/15675-
dc.descriptionA. Paniz-Mondolfi : A. H. Diwan : Y. Huttenbach Department of Pathology and Immunology, Baylor College of Medicine, Houston, TX, USA A. Paniz-Mondolfi (*) Departments of Biochemistry and Dermatopathology (SAIB/IVSS), Hospital Internacional& Universidad de Los Andes (ULA), Caracas/ Barquisimeto/Mérida, Venezuela e-mail: albertopaniz@yahoo.com R. Singh : B. A. Barkoh : R. Cason : J. Galbincea : R. Luthra Molecular Diagnostics Laboratory, The University of Texas MD Anderson Cancer Center, Houston, TX, USA G. Jour Department of Anatomic Pathology, Memorial Sloan Kettering Cancer Center, New York, NY, USA G. Jour Department of Pathology, Memorial Sloan Kettering, Cancer Center, New York, NY, USA M. Mahmoodi Department of Dermatopathology, Miraca Life Sciences Research Institute & Tufts University School of Medicine, Boston, MA, USA G. Benaim Laboratorio de Señalización Celular y Bioquímica de Parásitos, Instituto de Estudios Avanzados (IDEA), Caracas, Venezuela A. Paniz-Mondolfi Department of Pathology and Laboratory Medicine, Laboratorio de Bioquímica, (SAIB/IVSS), Hospital Internacional de Barquisimeto, Avenida Intercomunal Barquisimeto–Cabudare con Avenida La Montañita, Urb. Las Mercedes, Cabudare, Estado Lara, Venezuela.en_US
dc.description.abstractTo the editor: We thank Dr. Zidar and Dr. Gale for their timely and detailed comments on our paper [1]. Indeed, epithelial-mesenchymal transition (EMT) is a multifaceted process driven by an intricate network of molecular mechanisms which to date remain incompletely understood, due to the extraordinary complexity of their regulation [2]. These processes include changes in cell adhesion and shape, gene expression, and extensive cross-talk between signaling pathways that create complex biochemical networks [2, 3], of which the final result in cancer is the development of mesenchymal-like cells with enhanced capacity for invasion, migration, and dissemination[3].en_US
dc.language.isoenen_US
dc.publisherVirchows Archen_US
dc.relation.ispartofseriesVol. 466;No. 3 pp 359–360-
dc.subjectepithelial-mesenchymal transitionen_US
dc.subject(EMT)en_US
dc.subjectmolecular mechanismsen_US
dc.subjectcell adhesionen_US
dc.subjectgene expressionen_US
dc.subjectmesenchymal-like cellsen_US
dc.titleCutaneous carcinosarcoma and the EMT: to transition, or not to transition? That is the questionen_US
dc.typeArticleen_US
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