Inadequate epidermal homing leads to tissue damage in human cutaneous leishmaniasis.

Please use this identifier to cite or link to this item: https://saber.ucv.ve/jspui/handle/10872/13851

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DC Field	Value	Language
dc.contributor.author	Tapia, Félix J.	-
dc.contributor.author	Cáceres–Dittmar, Gisela	-
dc.contributor.author	Sánchez, Martín A.	-
dc.date.accessioned	2016-04-07T22:38:35Z	-
dc.date.available	2016-04-07T22:38:35Z	-
dc.date.issued	1994	-
dc.identifier.issn	0167-5699	-
dc.identifier.uri	http://hdl.handle.net/10872/13851	-
dc.description.abstract	Leishmaniasis is a model disease for the study of immunoregulatory mechanisms associated with host resistance and susceptibility: In this article, Felix Tapia and colleagues propose that defects in the signaling properties of the epidermis can result in the generation of either a chronic granulomatous response, which is unable to eliminate the parasite, or a proinflammatory mucocutaneous response and tissue damage.	es_VE
dc.language.iso	en	es_VE
dc.publisher	Immunology Today	es_VE
dc.relation.ispartofseries	Vol. 15;Nº 4	-
dc.subject	Leishmaniasis	es_VE
dc.subject	epidermis	es_VE
dc.subject	granulomatosa crónica	es_VE
dc.subject	parasite	es_VE
dc.subject	immunoregulatory mechanisms	es_VE
dc.title	Inadequate epidermal homing leads to tissue damage in human cutaneous leishmaniasis.	es_VE
dc.type	Article	es_VE
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